Joseph S Takahashi is a Japanese American neurobiologist and geneticist.
14 Facts About Joseph Takahashi
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Joseph Takahashi was elected to the National Academy of Sciences in 2003.
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Joseph Takahashi graduated from Richard Montgomery High School in Rockville, Maryland in 1970.
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Joseph Takahashi attended Swarthmore College and graduated with a degree in biology in 1974.
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Joseph Takahashi worked with Patricia DeCoursey at the University of South Carolina for a year after graduation and then applied to work with Michael Menaker at the University of Texas, Austin.
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Menaker ultimately moved to the University of Oregon where Joseph Takahashi received his neuroscience Ph.
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Joseph Takahashi was a postdoctoral fellow at the National Institute of Mental Health for two years under Martin Zatz before assuming a faculty position in Northwestern University's Department of Neurobiology and Physiology in 1983, where he held a 26-year tenure.
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Joseph Takahashi serves as a member of the Scientific Advisory Board of Hypnion Inc.
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Joseph Takahashi serves as a member of the editorial boards of Neuron, Physiological Genomics and Journal of Biological Rhythms.
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Joseph Takahashi's research has led to many developments in understanding how the circadian clock of mammals affects physiology and relationships with the environment.
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In 2000, Joseph Takahashi made what he calls one of his most significant contributions to the field, which was the cloning of the mutant tau gene identified in 1988 by Menaker and Martin Ralph.
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Since identifying the clock mutant in 1994, Joseph Takahashi has continued his research on this mutation and has applied it to studying clinical disorders, such as irregular sleep homeostasis and obesity.
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Joseph Takahashi's lab has continued use of this method in order to lead to discoveries of the role of the circadian clock in vision, learning, memory, stress, and addiction, among other behavioral properties.
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Joseph Takahashi and his colleagues proposed that FBXL3 is a target site for protein degradation on the CRY2 protein, which would explain relatively normal CRY2 protein levels.