Major Clinical depression significantly affects a person's family and personal relationships, work or school life, sleeping and eating habits, and general health.
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Major Clinical depression significantly affects a person's family and personal relationships, work or school life, sleeping and eating habits, and general health.
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Therapies associated with Clinical depression include interferons, beta-blockers, isotretinoin, contraceptives, cardiac agents, anticonvulsants, antimigraine drugs, antipsychotics, and hormonal agents such as gonadotropin-releasing hormone agonist.
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Depression occurring after giving birth is called postpartum Clinical depression and is thought to be the result of hormonal changes associated with pregnancy.
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Seasonal affective disorder, a type of Clinical depression associated with seasonal changes in sunlight, is thought to be triggered by decreased sunlight.
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Pathophysiology of Clinical depression is not completely understood, but current theories center around monoaminergic systems, the circadian rhythm, immunological dysfunction, HPA-axis dysfunction and structural or functional abnormalities of emotional circuits.
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HPA-axis abnormalities have been suggested in Clinical depression given the association of CRHR1 with Clinical depression and the increased frequency of dexamethasone test non-suppression in people who are depressed.
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Development of mental health services is minimal in many countries; Clinical depression is viewed as a phenomenon of the developed world despite evidence to the contrary, and not as an inherently life-threatening condition.
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Most-studied form of psychotherapy for Clinical depression is CBT, which teaches clients to challenge self-defeating, but enduring ways of thinking and change counter-productive behaviors.
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People with major Clinical depression are less likely to follow medical recommendations for treating and preventing cardiovascular disorders, further increasing their risk of medical complications.
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Major Clinical depression is about twice as common in women as in men, although it is unclear why this is so, and whether factors unaccounted for are contributing to this.
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The risk of major Clinical depression is increased with neurological conditions such as stroke, Parkinson's disease, or multiple sclerosis, and during the first year after childbirth.
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Major Clinical depression was the leading cause of disease burden in North America and other high-income countries, and the fourth-leading cause worldwide as of 2006.
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Term Clinical depression itself was derived from the Latin verb, meaning "to press down".
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Clinical depression theorized that objective loss, such as the loss of a valued relationship through death or a romantic break-up, results in subjective loss as well; the depressed individual has identified with the object of affection through an unconscious, narcissistic process called the libidinal cathexis of the ego.
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Clinical depression emphasized early life experiences as a predisposing factor.
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New definitions of Clinical depression were widely accepted, albeit with some conflicting findings and views.
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Similarly, Hungarian-American psychiatrist Thomas Szasz and others argue that Clinical depression is a metaphorical illness that is inappropriately regarded as an actual disease.
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Some well-known contemporary people with possible Clinical depression include Canadian songwriter Leonard Cohen and American playwright and novelist Tennessee Williams.
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Social stigma of major Clinical depression is widespread, and contact with mental health services reduces this only slightly.
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